Exercise by releasing a protein hormone helps prevent memory loss in Alzheimer's disease January 15, 2019 Source: Science and Technology Daily Author: Zhang Meng Ran According to a recent study published online by the British journal Nature Medicine, a joint team between the United States and Brazil found that a motor-induced protein hormone is closely related to the progression of Alzheimer's disease, which prevents Alzheimer's disease. Loss of disease memory, the therapeutic strategy developed thereby, may pave the way for amelioration of cognitive decline associated with human disease. Since the pathogenesis is still unclear, people cannot fundamentally prevent or delay the progression of Alzheimer's disease. Exercise is known to benefit many systems, including the health of the brain. Past research has shown that physical activity may enhance cognitive ability and may even delay the progression of neurodegenerative diseases such as Alzheimer's disease. During exercise, a messenger protein called irisin is released from muscle tissue, enters the blood circulation, and acts on distant targets. A joint research team between Columbia University and the Federal University of Rio de Janeiro in Brazil found that in a mouse model of Alzheimer's disease, irisin enhances the cognitive effects of exercise. Moreover, the expression level of iris and its protein precursor FNDC5 in the brain of Alzheimer's patients was lower than that of healthy controls. The researchers also observed this phenomenon in a mouse model of Alzheimer's disease. The team also found that just as reducing the gene expression of FNDC5/Irisin induces learning and memory loss and restores its expression, it can be reversed in a mouse model. Furthermore, in the mouse model of Alzheimer's disease, when the signaling of FNDC5/Irisin in the brain or periphery is blocked, the cognitive effect of sports disappears. This finding may be significant for conquering Alzheimer's disease and will open the door to designing new therapeutic strategies to alleviate cognitive decline in patients. Further research is needed to understand more accurately how the iris has entered the brain and interacted with it to accurately assess the similar cognitive effects of irisin in the human body.
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