Release date: 2015-08-05 Source: Bio Valley
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Why do people get older? For humans, how to live forever is really a fascinating problem. But so far there is no satisfactory answer. Recently, scientists from Berlin, Germany, conducted a study that took another step in solving the mystery of human aging. They found that the endoplasmic reticulum region of aging cells loses oxidative motility, causing many proteins to fail to undergo normal folding modifications. The discovery of a mature protein with normal function is of great significance for understanding the process of cellular senescence. Relevant research results were published in the international academic journal EMBO Journal.
Each cell is composed of different cellular components, and the endoplasmic reticulum is an important organelle in the cell. Some secreted proteins, such as antibodies produced by insulin or immune cells, need to be processed and matured in the oxidative environment of the endoplasmic reticulum. Therefore, redox balance is important for maintaining the normal maturation process of immature proteins inside the endoplasmic reticulum.
In this study, scientists first discovered that the endoplasmic reticulum loses oxidative motility in senescent cells, altering the redox balance, leading to a decrease in disulfide bond formation, and the formation of disulfide bonds within proteins is important for the formation of correct protein folding. effect. The decrease in disulfide bond forming ability directly causes many proteins to fail to mature normally and become unstable.
Researchers have pointed out that previous studies have found that misfolded proteins increase during aging, but what changes have occurred in the endoplasmic reticulum remains unclear, and this study reveals for the first time the endoplasmic reticulum inside the aging process. The change in redox balance is an important reminder for understanding why the secreted proteins in the elderly and the body are stressed and lose normal function, and also to understand Alzheimer's disease, Parkinson's disease Important information is provided for the causes of senile diseases such as Huntington's disease.